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The mechanisms likely involve immune modulation (microglia in ALS can adopt a neurodegenerative phenotype that might be restrained by microbial signals) and metabolic support (providing nutrients like nicotinamide or SCFAs to neurons and glia) [214, 218]

372 In addition, in terms of the major mechanism of hyperuricemia induced commodities, oxidative stress, inflammatory signaling pathway and immune response are involved in this process, which mainly lead to cell apoptosis and endothelial dysfunction

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